List of publications using KU 0058948 HCl (Axon 2001) purchased from Axon Medchem

(Total 18 publication citations listed; updated up to March 2020)

2019
Martinez-Macias, M. I., Moore, D. A., Green, R. L., Gomez-Herreros, F., Naumann, M., Hermann, A., ... & Caldecott, K. W. (2019). FUS is part of the cellular response to topoisomerase-induced DNA breaks and transcriptional stress. Life Science Alliance. 

Martinez-Macias, M. I., Moore, D. A., Green, R. L., Gomez-Herreros, F., Naumann, M., Hermann, A., ... & Caldecott, K. W. (2019). FUS (fused in sarcoma) is a component of the cellular response to topoisomerase I–induced DNA breakage and transcriptional stress. 
Life science alliance2(2). 

Nie, Y., Li, Y., Li, X., Wilson, A. F., & Pang, Q. (2019). The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance. Stem cell research & therapy, 10(1), 114.
https://stemcellres.biomedcentral.com/articles/10.1186/s13287-019-1206-0

Mesman, R. L., Calléja, F. M., Hendriks, G., Morolli, B., Misovic, B., Devilee, P., ... & Vreeswijk, M. P. (2019). The functional impact of variants of uncertain significance in BRCA2. Genetics in Medicine, 21(2), 293-302.
https://www.nature.com/articles/s41436-018-0052-2/

Wijenberg, L. (2019). PARP inhibitor resistance in BRCA1/2 mutated tumors (RuG Doctoral dissertation).
http://fse.studenttheses.ub.rug.nl/21252/1/mBMS_2019_WijenbergL.pdf

2018
Boetefuer, E. L., Lake, R. J., Dreval, K., & Fan, H. Y. (2018). Poly (ADP-ribose) polymerase 1 (PARP1) promotes oxidative stress–induced association of Cockayne syndrome group B protein with chromatin. Journal of Biological Chemistry, 293(46), 17863-17874.
https://www.jbc.org/content/293/46/17863.full

Boetefuer, E. L. (2018). Mechanisms Underlying Oxidative Stress-Induced Chromatin Association Of Cockayne Syndrome Protein B (csb). UPENN PhD Dissertation.
https://repository.upenn.edu/edissertations/2766/

Nagle, P. W., Hosper, N. A., Barazzuol, L., Jellema, A. L., Baanstra, M., van Goethem, M. J., ... & Coppes, R. P. (2018). Lack of DNA damage response at low radiation doses in adult stem cells contributes to organ dysfunction. Clinical Cancer Research, clincanres-0533.
* KU-55933 and KU-0058948 from Axon Medchem
 
Mesman, R. L., Calléja, F. M., Hendriks, G., Morolli, B., Misovic, B., Devilee, P., ... & Vreeswijk, M. P. (2018). The functional impact of variants of uncertain significance in BRCA2. Genetics in Medicine, 1.
 
2017
Hoch, N. C., Hanzlikova, H., Rulten, S. L., Tétreault, M., Komulainen, E., Ju, L., ... & Staras, K. (2017). XRCC1 mutation is associated with PARP1 hyperactivation and cerebellar ataxia. Nature, 541(7635), 87.
 
Mateos-Gomez, P. A. (2017). Deciphering the Role of Alternative Non-Homologous End Joining (Alt-NHEJ) DNA Repair in Breast Cancer. New York University School of Medicine New York United States.
 
Meijers-Heijboer, J. W., Waisfisz, Q., & Wolthuis, R. M. ANALYSIS of THE fANCoNI ANEmIA REPAIR PATHWAY IN BREAST CANCER IDENTIfIES RECQL5 AmPLIfICATIoN AS A mEDIAToR of DNA CRoSSLINKER SENSITIvITY. HEREDITARY BREAST CANCER Of genes and therapy, 81.
 
Day, T. A., Layer, J. V., Cleary, J. P., Guha, S., Stevenson, K. E., Tivey, T., ... & Root, D. E. (2017). PARP3 is a promoter of chromosomal rearrangements and limits G4 DNA. Nature Communications, 2017 8, 15110.
 
Skvarova Kramarzova, K., Osborn, M. J., Webber, B. R., DeFeo, A. P., McElroy, A. N., Kim, C. J., & Tolar, J. (2017). CRISPR/Cas9-Mediated Correction of the FANCD1 Gene in Primary Patient Cells. International Journal of Molecular Sciences, 18(6), 1269.
 
Gilmore, J. M., Sardiu, M. E., Groppe, B. D., Thornton, J. L., Liu, X., Dayebgadoh, G., ... & Florens, L. (2016). Wdr76 co-localizes with heterochromatin related proteins and rapidly responds to dna damage. PloS one, 11(6), e0155492.
 
Hanzlikova, H., Gittens, W., Krejcikova, K., Zeng, Z., & Caldecott, K. W. (2016). Overlapping roles for PARP1 and PARP2 in the recruitment of endogenous XRCC1 and PNKP into oxidized chromatin. Nucleic acids research, 45(5), 2546-2557.
 
Ectopic expression of RNF168 and 53BP1 increases mutagenic but not physiological non-homologous end joining.
Zong, D., Callén, E., Pegoraro, G., Lukas, C., Lukas, J., & Nussenzweig, A. 
Nucleic Acids Res. 2015 May 26;43(10):4950-61. doi: 10.1093/nar/gkv336. Epub 2015 Apr 27.

Mammalian polymerase θ promotes alternative NHEJ and suppresses recombination.
PA Mateos-Gomez, F Gong, N Nair, KM Miller, E Lazzerini-Denchi & A Sfeir
Nature 2015, 518, 254–257. doi:10.1038/nature14157
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